Facts: 1. A carboxyl group is required to attach the fatty acid chain to the glycerol molecule. 2. By the time fatty acids are converted to DHA, we are only able to use about 5% of it. 3. PUFA's are very effective at neutralizing super oxides (aka free radicals) Questions: 1. Are the Mylan Sheaths made up largely of Omega-3 and Omega-6 fatty acids, or are they made up of "Saturated" fatty acids. 2. Why are the superoxides made (what biological process produces them and why is it necessary).
1. Part of Anthony's video talks about the anatomy of the neurons. He points out the there are different types of neurotransmitters with different functions. This connects with my video about synaptic transmission where we can go deeper into the effects of PUFAs on the different parts of the neurons not only the axon part. 2. a) Why does super-oxide targets the PUFAs? How does this process happen? That might be an importnat thing to understand to fix this issue. b) What effects does PUFAs has on different neurotransmitters?
Some things to look up and elaborate on: - What are ester linkages? We actually take the methyl esters and put those through the GC. - What is the mechanism by which FAs help decrease the fibrosis of cardiac muscle? - What is the mechanism by which FAs help increase muscle growth? - How do FAs affect neurotransmitters? - What is the structure (Lewis dot diagram) of a superoxide? - Show the structure of a free radical. What makes it so unstable/highly reactive?
Overall, a good job Anthony. A great summary of what you've learned so far!
Facts:
ReplyDelete1. A carboxyl group is required to attach the fatty acid chain to the glycerol molecule.
2. By the time fatty acids are converted to DHA, we are only able to use about 5% of it.
3. PUFA's are very effective at neutralizing super oxides (aka free radicals)
Questions:
1. Are the Mylan Sheaths made up largely of Omega-3 and Omega-6 fatty acids, or are they made up of "Saturated" fatty acids.
2. Why are the superoxides made (what biological process produces them and why is it necessary).
- Myelin sheaths
Delete1. Part of Anthony's video talks about the anatomy of the neurons. He points out the there are different types of neurotransmitters with different functions. This connects with my video about synaptic transmission where we can go deeper into the effects of PUFAs on the different parts of the neurons not only the axon part.
ReplyDelete2.
a) Why does super-oxide targets the PUFAs? How does this process happen? That might be an importnat thing to understand to fix this issue.
b) What effects does PUFAs has on different neurotransmitters?
Some things to look up and elaborate on:
ReplyDelete- What are ester linkages? We actually take the methyl esters and put those through the GC.
- What is the mechanism by which FAs help decrease the fibrosis of cardiac muscle?
- What is the mechanism by which FAs help increase muscle growth?
- How do FAs affect neurotransmitters?
- What is the structure (Lewis dot diagram) of a superoxide?
- Show the structure of a free radical. What makes it so unstable/highly reactive?
Overall, a good job Anthony. A great summary of what you've learned so far!